As much as I believe it possible, that video is a fake. I would, no matter my position nor responsibility, sit through a presentation by such a poor speaker, no matter how valid nor helpful his theory. Nor would a professional interrupt nor speak in such a manner while aware they are being recorded (And if they're in the pentagon, and WORK in the pentagon, they're MORE than aware there are recording devices on them)
P.S. I'm going to include a link to the user's "channel" who originally posted the video linked above. Take a look at some of the other videos, you'll see. Also, the fact that youtube hasn't pulled the video down do too infringements says a lot.
Edited by Darkorpse (07/20/1101:48 AM)
Sic is est nex, Ego volo magis
Caution: Messages may contain content not suitable for some viewers
Loc: Kamloops BC Canada
Video at the bottom is opinionated somewhat...
August 22, 2011 DRACOs: New Antivirals Against Pretty Much Everything? Posted by Derek
I've been meaning to write about this paper from the RIder group at MIT's Lincoln Labs, which shows some very interesting approaches to killing off a wide variety of viruses. They've dubbed these new agents DRACOs, for Double-stranded RNA Activated Caspase Oligimerizers, which is certainly one of those acronyms with a lot packed into it.
So now to unpacking it. The first key point is the double-stranded RNA (dsRNA) part. For a long time, that was thought to be a form that isn't wasn't found in human cells (as opposed to single-stranded stuff). We now know that short dsRNAs (up to twenty-odd base pairs) are part of human biology, but viruses produce much longer strands of it during their replication process - or, more accurately, they hijack human cellular machinery to produce it. (Viruses, as a rule, don't do anything for themselves that they don't absolutely have to).
Naturally enough, cells have evolved ways to recognized long dsRNAs as a sign of infection - there's a whole list of proteins that recognize these things and bind to them. Some of them inhibit its downstream processing directly, by just hanging on and gumming up the works, while others set off responses further downstream. One of those is apoptosis, programmed cell death, a brutal but effective fall-on-your-sword pathway that gets initiated by all sorts of unfixable cellular problems. (When a cell's internal controls give a "Fatal Error" message, it's taken literally). And naturally enough, viruses have evolved ways to try to evade these defenses, both by targeting the dsRNA detection proteins and by inhibition of apoptosis pathways. (As a side note, it's always been interesting to untangle these counter-counter-countermeasure situations whenever a new cellular pathway relating to infection is worked out. You find, invariably, that hundreds of millions of years of evolutionary pressure have built up crazily elaborate frameworks around all of them).
This approach tries to speed up the dsRNA-means-apoptosis connection. A DRACO turns out to be a good-sized protein with two functions: one end recognizes and binds to dsRNA, and the second contains a signal to induce apotosis. If multiple copies of the DRACO protein stick to the same viral dsRNA strand, that should be enough to initiate cell death and interrupt the viral replication process. The team tried out a whole range of possibilities for both those functional domains, with the best (so far) using either Protein Kinase R (PKR) or RNAaseL domains to recognize viral RNA and an Apaf caspase recruitment domain for apoptosis signaling. Another key modification was the addition of a PTD (protein transduction domain) tag, which allows large proteins like these entry into cells through active transport. (Cells only take in whole proteins through gatekeeping transport mechanisms; otherwise they just sort of bounce off - this effect was confirmed with DRACOs that lacked the PTD tags).
So, basically, this is the sort of protein that you might expect evolution to stumble onto eventually, but now the connecting line has been drawn by hand instead. It's worth noting at this point, though, that this general idea has occurred to others before: here's a paper from Boston University trying the same sort of strategy. That one was published online in 2009, but didn't make it to print until May of this year, which makes you wonder if that's a typical delay for that journal (FASEB J.) or not. It's also worth noting that, for whatever reason, this new MIT paper does not cite the one from BU.
How did they work? Pretty well. The PTD tags did what they were supposed to, taking the proteins into cells rapidly. Once inside, the DRACOs themselves hung around for several days before being degraded, which is another big hurdle. And they did indeed protect against infection by an impressively wide range of viruses in cell culture: rhinovirus, encephalomyelitis, adenoviruses, arenaviruses, bunyaviruses, flaviviruses, reovirus, and flu. A lot of nasty pathogens fall into those bins.
Loc: In Nanna Bijou's Shadow
Read Horowitz Emerging Viruses this will change your minds on the vaciniation issue. here's a link to the book review. it was published over ten years ago so you can get a copy from abebook.com for a few bucks, but it will reorient your reality.
Loc: Kamloops BC Canada
Based on the best-selling book by the same name, Dr. Len Horowitz presents the definitive exploration into the origins of the AIDS and Ebola viruses. Claims that these "emerging viruses" naturally evolved and then jumped species from monkey to man are laughable in light of the documented evidence in this extraordinary lecture. These bizarre germs were undoubtedly man-made creations, accidentally or intentionally transmitted via tainted vaccines in the U.S. and Africa. The genocidal theory of AIDS is meticulously explored within the social and political context of this stormy period of American military science. This lecture exposes the potential motives of administrations responsible for this atrocity, and the methods they continue to use to deceive and kill unsuspecting populations.
From the article Dark quoted: "This is how vaccine dogma works, though. The religion of vaccines does not have to offer any solid proof that a vaccine works, or that it is even safe. A vaccine can even cause the very thing it is touted as preventing, and vaccine apologists will say that it works and that it is necessary. And now in this case, these same psychopaths are suggesting that young babies be starved of real nutrition in order to improve the effectiveness of a vaccine."
I don't know who wrote this, nor their background in vaccine production and/or testing, but it is so far from the truth, it's farcical.
One of my first jobs was to work with agricultural vaccines, testing for purity (of live vaccines), potency, safety and efficacy. Now, I am speaking from my experience as a Canadian scientist, so this may not apply down south (although I'd be shocked if it wasn't the same). The lab I helped to build back in the early eighties had only one job, as preciously described. If a vaccine did not pass all tests to ensure it was not contaminated, was safe, was not too weak (e.g. the so-called holistic vaccines) or did not offer protection from the target antigen(s), it was not licensed in this country. I know my colleagues at Health Canada were/are doing similar testing on human vaccines.
As for vaccines causing the very diseases they are meant to stop, the author uses a blanket statement which strongly implies all vaccines can be guilty of this. Nope, not even close. The majority of vaccines do NOT contain live, attenuated target organisms, but rather they incorporate only specific antigens (proteins) from the disease causing microbes. By the way, attenuated means weakened. The target viruses or bacteria are chemically or physically treated to render them harmless, yet still alive. This gives the vaccine a much greater ability to stimulate excellent antibody production, which is the point of it all.
I have never come across an attenuated vaccine which was blamed for causing the disease it was meant to prevent - after more than a decade being involved in their testing.
It's a jeep. If I'd wanted a hummer, I would have called your sister.