Hey Subcool.

Tried some african buzz fully cured and that shit gives a crazy headrush. pure sativas r the best man, can smell it through the freezer bags. Hey subcools back

I bet that THCV has a lot to
do with the high of that strain.

If people want CBD it's a bit low.

Some disagree on the THCV thing.
Here is more
from an IC thread (sam skunkman).

http://www.cannabisseur.org/Tetrahydrocannabivarin

Tetrahydrocannabivarin

"THCV is the propyl homologue of THC. In the aromatic ring the usual five-carbon pentyl is replaced by a short three-carbon propyl chain. The propyl cannabinoids have so far been found in some varieties originating from Southeast and Central Asia and parts of Africa. What are considered some very potent marijuana varieties contain propyl cannabinoids. In one study, THCV made up to 48.23 percent (Afghanistan strain) and 53.69 percent (South Africa) of the cannabinoids found (136). We've seen no reports on its activity in humans. From animal studies it appears to be much faster in onset and quicker to dissipate than THC (181). It may be the constituent of one- or two-toke grass, but its activity appears to be somewhat less than that of THC."[1]

"THCV is a THC antagonist, it prevents you from getting high on THC, like CBD does if CBD is taken in large doses before the THC. I also smoked herbal cannabis with 1% THCV/10%THC, 5%THCV/5% THC, 10%THCV/1% THC, the rest of the Cannabinoids being absent. And you do not want THCV, it ruins the high... I do know why tropical varieties can sometimes have an amazing fast up high. It has nothing to do with THCV. But if you do not believe me do find some variety with a small amount of THCV and self the female a few generations and you will end up with a THCV dominate plant, if you have a GC or other analytical method to find it."[2]

"Thai, Asian, Afghan, and African strains have THCV in very small amounts"[3]

"THCV bound to the CB1 receptor with similar affinity as�9-THC, it had no effects when administered alone, but antagonized the antinociceptive effects of �9-THC when both drugs were given in combination" [4]

from Wiki:

Natural occurrence

THCV is found in largest quantities in Cannabis sativa subsp. sativa strains. Some varieties that produce propyl cannabinoids in significant amounts, over five percent of total cannabinoids, have been found in plants from South Africa, Nigeria, Afghanistan, India, Pakistan and Nepal with THCV as high as 53.69% of total cannabinoids.[1] They usually have moderate to high levels of both THC and Cannabidiol (CBD) and hence have a complex cannabinoid chemistry representing some of the world's most exotic cannabis varieties.[2]
[edit] Pharmaceutical properties

It has been shown to be a CB1 receptor antagonist, i.e. blocks the effects of THC.[3] In 2007 GW Pharmaceuticals announced that THCV is safe in humans in a clinical trial and it will continue to develop THCV as a potential cannabinoid treatment for type 2 diabetes and related metabolic disorders, similar to the CB1 receptor antagonist rimonabant.[4]

THCV can also be found in African wild Dagga aka "lions tail" and can also be purchased online or at a herbal supply shop

http://en.wikipedia.org/wiki/Leonotis_leonurus

http://www.erowid.org/plants/leonotis_leonurus/

http://yourbrainonbliss.com/Blog/?p=1383


New British and Spanish research on one of cannabis’ cannabinoids show its great potential for treating Parkinson’s disease. The cannabinoid is the lesser known but hugely interesting THCV, aka Delta-9-tetrahydrocannabivarin. The molecule is present to varying decrees in different strains of cannabis, from trace amounts to a hefty proportion.

Unlike your own body’s cannabinoid anandamide, or its phyto(plant based)-cannabinoid cousin, THC, THCV does not activate CB1 receptors in your endocannbinoid regulatory system. Activation of these CB1 receptors, found mainly on nerve cells, is responsible for most of THC’s psychoactive effects and medical benefits. THC also activates CB2 receptors, found more on immune cells and thought responsible for some of cannabis’ beneficial effects on some autoimmune disorders. Like THC, THCV also binds with and activates these CB2 receptors. Like THC, THCV is a powerful antioxidant, capable of sopping up cell-killing free radicals. Unlike THC, THCV does not activate CB1 receptors. Instead, it blocks (serves as an antagonist to) the activation of the CB1 system. It may play a major role in future treatments of cardiometabolic diseases and obesity.

The International Association of Cannabinoid Medicine reported the research as follows:

Parkinson’s disease
Spanish and British researchers investigated the effects of Delta-9-
tetrahydrocannabivarin (THCV) in an animal model of Parkinson’s
disease. They concluded that “given its antioxidant properties and
its ability to activate CB2 but to block CB1 receptors, Delta-9-THCV
has a promising pharmacological profile for delaying disease
progression in PD and also for ameliorating parkinsonian symptoms.”
(Source: García C, et al. Br J Pharmacol. 2011 Feb 16. [in press]).

snip